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VLDLR

VLDL receptor

very-low-density-lipoprotein receptor (VLDLR) is a transmembrane lipoprotein receptor of the low-density-lipoprotein (LDL) receptor family. VLDLR shows considerable homology

Uner Tan syndrome

gene (VLDLR) is located in this region, which is involved in the migration of neuroblasts within the brain. Ozcelik found mutations in the VLDLR gene in

Low-density lipoprotein receptor gene family

seven members of the LDLR family in mammals, namely: LDLR VLDL receptor (VLDLR) ApoER2, or LRP8 Low density lipoprotein receptor-related protein 4 also

VLDLR-associated cerebellar hypoplasia

VLDLR-associated cerebellar hypoplasia (VLDLRCH) is a rare autosomal recessive condition caused by a disruption of the VLDLR gene. First described as a

Reelin

apolipoprotein E receptor 2 (ApoER2) and very-low-density lipoprotein receptor (VLDLR), were discovered by Trommsdorff, Herz and colleagues, who initially found

Low-density lipoprotein receptor-related protein 8

migration and postnatal long-term potentiation. Another LDL family receptor, VLDLR, also interacts with reelin, and together these two receptors influence

Norman–Roberts syndrome

The other is VLDLR-associated cerebellar hypoplasia, which is caused by a mutation in the gene coding for one of the reelin receptors, VLDLR.[citation needed]

DAB1

intracellular part of the Reelin very low density lipoprotein receptor (VLDLR) and apoE receptor type 2 (ApoER2) and becomes tyrosine-phosphorylated following

Cajal–Retzius cell

signaling pathway, including the very low density lipoprotein receptor (VLDLR), the apolipoprotein E receptor type 2 (ApoER2), and the cytoplasmic adapter

Reeler

role of downstream molecules. The examples include: Double knockout of VLDLR and ApoER2 receptors; Double knockout of Src and Fyn kinases. Cre-loxP recombination